Research in our laboratory focuses on determining: 1) the molecular and cellular mechanisms underlying the brain signal for ovulation; 2) developmental mechanisms responsible for sex differences in the brain; 3) how developmental exposure to environmental contaminants like dioxins and estrogenic substances produces neural dysfunctions in adulthood.
The mechanisms through which a non-classical progesterone signaling molecule, progesterone receptor
component 1, suppresses neuroimmune responses to proinflammatory cytokines in the etiology and
progression of neurodegenerative diseases.
- Mechanisms through which CUG binding protein 2 directs dendritic spine development, a process important for functions ranging from learning and memory to ovulation.
- The effects of dioxin-disruption of CUG binding protein 2 during early life on the development of neurodegenerative diseases.
Learn more at www.vasci.umass.edu/research-faculty/sandra-l-petersen
- BS Biology, Rutgers University, 1977
- MS Neuroendocrinology, Oregon State University, 1980
- PhD Neuroendocrinology, Oregon State University, 1984
- Postdoctoral training, University of Maryland School of Medicine, 1984-1988